By Elisabeth von der Lohe
Take benefit of new insights into prevention, analysis and therapy of heart disorder in ladies - to the advantage of your sufferers. Designed to optimize your day-by-day perform, this ebook supplies lucid, updated solutions to various questions:
- What are the dangers and merits of hormone alternative therapy?
- What are the main chance components for heart affliction in women?
- What are the main applicable diagnostic checks in women?
- that's the higher remedy choice for the lady with myocardial infarction - lysis or fundamental angioplasty?
Read or Download Coronary Heart Disease in Women: Prevention - Diagnosis - Therapy PDF
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Reap the benefits of new insights into prevention, prognosis and remedy of heart disorder in ladies - to the advantage of your sufferers. Designed to optimize your day-by-day perform, this booklet offers lucid, updated solutions to various questions:- What are the dangers and merits of hormone alternative treatment?
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Extra resources for Coronary Heart Disease in Women: Prevention - Diagnosis - Therapy
67 References . . . . . . . . . . . . . 77 E. von der Lohe, Coronary Heart Disease in Women © Springer-Verlag Berlin Heidelberg 2003 48 Chapter 4 · Stable Angina Definition and Pathophysiology of Angina Pectoris Angina is a clinically defined syndrome rather than a disease and does not always imply the presence of coronary artery disease; however, angina pectoris is the most common manifestation of coronary artery disease and in this situation symptoms are due to myocardial ischemia.
It increases fibrinolytic activity. References Chang WC, Nakao J, Orimo H, Murota SI (1980) Stimulation of prostacyclin biosynthetic activity by estradiol in rat aortic smooth muscle cells in culture. Biochim Biophys Acta 619:107–118 Collins P, Rosano G, Sarrel PM et al. (1995) Estrogen attenuates acetylcholine-induced coronary arterial constriction in women but not in men with coronary heart disease. Circulation 92:24–30 Gebara OC, Mittleman MA, Sutherland P et al. (1995) Association between increased estrogen status and increased fibrinolytic potential in the Framingham Offspring Study.
Leads to changes in the release of vasoactive substances, to increased uptake of lipoproteins by the intima, and to expression of glycoproteins on the endothelium cell surface which serves as adhesion molecules for monocytes and T-lymphocytes. As a result of the deposit of lipids, the monocytes change into foam cells which subsequently form fatty streaks together with lymphocytes. Intermediate lesions occur after the proliferation of SMCs and microphages, and complex so-called fibrous plaques form after the accumulation of lipids in the cells and connective tissue matrix.
Coronary Heart Disease in Women: Prevention - Diagnosis - Therapy by Elisabeth von der Lohe