By Philippa J. Easterbrook MB BChir BSc(Hons) FRCP DTM&H MPH

ISBN-10: 0443073260

ISBN-13: 9780443073267

Written for applicants sitting their MRCP half 1 exam, this revision makes a speciality of the habitual topics which arise within the questions. The ebook additionally contains a bankruptcy on scientific pharmacology (which on my own money owed for as much as 30% of the questions), facets of drug-induced disorder and drug interactions. eventually there's a bankruptcy on statistics and epidemiology which is never lined in different texts, yet is frequently incorporated within the exam.

  • Helps MRCP half 1 applicants arrange for and cross their exam.
  • Addresses an more and more very important subject within the exam.
  • Addresses a subject that's very important to passing the examination, yet which such a lot applicants are poorly ready for.
  • Covers all of the correct uncomplicated technology topics plus contains medical pharmacology.
  • Is of use to applicants learning for different postgraduate assessments comparable to PLAB, USMLE and MRCPCH.
  • Is the 1st e-book of its sort within the club marketplace and is now considered as crucial for examination preparation.

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Extra resources for Basic Medical Sciences for MRCP Part 1

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Closely associated loci in the same chromosome are linked if genes at these loci segregate together more than 50% of meioses. The lod score is a mathematical indication of the relative likelihood that two loci are linked. A lod score >3 is confirmation of linkage. 11 Ethnic groups and genetic disease Population with high frequency Associated disease Scandinavians a,-Antitrypsin and lecithin cholesterol acyl transferase (LCAT) deficiency, congenital nephrotic syndrome Cystic fibrosis Tay-Sachs disease, Gaucher's disease Familial Mediterranean fever Congenital adrenal hyperplasia and pseudocholinesterase deficiency ~-Thalassaemia, glucose-6-phosphate dehydrogenase deficiency and familial Mediterranean fever Glucose-6-phosphate dehydrogenase deficiency Haemoglobinopathics especially sickle cell disease, a - and Northern Europeans Ashkenazi Jews Sephardic Jews and Armenians Eskimos Mediterranean races Chinese Africans ~-thalassaemias South African whites Porphyria variegata 31 ~ Heterozygotic advantage • When a serious autosomal recessive disorder has a relatively high incidence in a large population , this is likely to be due to heterozygote advantage.

Each cell typically expresses 16 000-20 000 genes. Cf) • Expression is regulated by transcription factors ; proteins which bind to DNA at the 5' end of genes (the promoter) and initiate transcription of genes (Fig . 17). • Some genes have a fundamental biological role and will be expressed in all cells at all times ('housekeeping genes'). ~ () 1-- w z w (9 Promoter Exons Fig. 17 Promoter, introns and exons. THE SOMATIC EVOLUTION OF CANCER • Cancer cells are a clonal population of cells. The malignant phenotype arises from the accumulation of mutations in multiple genes.

Psittaci C. trachomatis C. } 0_J 0 en 0 a: Characteristic Exotoxin Produced by: Certain Gram +ve and -ve bacteria Gram - ve bacteria Composed of: Secreted polypeptide Stability Endotoxin Heat labile Lipid A in lipopolysaccharide in cell wall Heat stable Antigenicity Highly antigenic. Toxoids Poorly antigenic Biological actions Inhibits protein synthesis LPS molecule induces TNFa Clinical effects Various i Production of cAMP Anthrax toxin Cholera toxin: secretory diarrhoea E. coli enterotoxin: secretory diarrhoea Pertussis toxin : fluid and electrolyte loss, mucus secretion in respiratory tract Inhibition of protein synthesis Diphtheria toxin: cell death P.

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Basic Medical Sciences for MRCP Part 1 by Philippa J. Easterbrook MB BChir BSc(Hons) FRCP DTM&H MPH

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